In the era of enhanced antenatal ultrasound and fetal echocardiogram programs, the majority of these patients are diagnosed antenatally - Prashant K. Minocha
image by: Maita Fabella Mojado
In its place is fibrous tissue that closes it, preventing the passage of oxygen-poor blood from the right atrium to the right ventricle. The right ventricle is often small (hypoplastic) due to reduced blood flow as early as fetal development.
In tricuspid atresias that are not life-threatening, there is at least one defect in the wall (septum) separating the two atria or the septum separating the two ventricles (usually both). This makes it possible for non-oxygenated blood to mix with oxygenated blood.
Tricuspid atresia is manifested by cyanosis (bluish color of the child’s skin) due to the mixing of non-oxygenated blood with oxygenated blood from the left ventricle to the whole body. Some children are moderately cyanotic and can survive long enough; others need early surgery because the body’s oxygen demands are not being met.
Tricuspid atresia (TA) is a cyanotic congenital heart defect characterized by the complete agenesis of the tricuspid valve (TV). That is, there is no communication between the right atrium and ventricle. There are several subtypes of this disease with varied clinical presentations based on the degree of pulmonary blood flow.
Tricuspid atresia is the 3rd most common cyanotic congenital heart disease with a prevalence of 1 per 10,000 births. It accounts for 1-3% of all congenital heart anomalies.
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